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In vivo experiments clearly show anatomically selective neurochemical outcomes of modafinil on monoaminergic programs (de Saint Hilaire et al 2001; Ferraro et al 2002), and, notably, whilst modafinil boosts TMN fos expression (Scammell et al 2000) and HAergic tone it can be unable to exert this effect when administered specifically into the TMN (Is

copyright also blocks sodium channels, thereby interfering With all the propagation of motion potentials;[114][72] Therefore, like lignocaine and novocaine, it functions as a local anesthetic. In addition, it features within the binding sites on the dopamine and serotonin sodium dependent transport area as targets as different mechanisms from its r